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Further reading of "Phagocytic Clearance in Neurodegeneration" revealed that AB (which form?) causes an inflammatory response in microglia that is responsible for neurodegeneration, while upregulating anti-inflammatory cytokines could help increase phagocytic activity and reduce neurodegeneration. Could that be an added/alternate approach? How would we test if it is working?
Looking into:
TGF-β1 promotes microglial amyloid-β clearance and reduces plaque burden in transgenic mice TGF-beta1 promotes microglial AB clearance.pdf
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