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Here are a couple of naturally occurring AB receptors to look into, in case LilrB2/PirB don't behave how we want them to. Paper: Phagocytic clearance in Neurodegeneration (and AD).pdf (I'm guessing there's a better way to do this?)
Not sure how much we want to use them given that they’re naturally present in all relevant cell types in the brain. I have a feeling that FPRL1 and RAGE would be most promising, though I haven't done any research on any of them yet.